Spontaneous remission of genetic, apparent primary, FSGS presenting with nephrotic syndrome challenges traditional notions of primary FSGS
Author:
Publisher
Springer Science and Business Media LLC
Subject
Nephrology
Link
https://link.springer.com/content/pdf/10.1007/s40620-020-00837-7.pdf
Reference5 articles.
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2. Pollak M (2016) Genetics of familial FSGS. Semin Nephrol 36(6):467–472. https://doi.org/10.1016/j.semnephrol.2016.09.010
3. Armstrong ME, Thomas CP (2019) Diagnosis of monogenic chronic kidney diseases. Curr Opin Nephrol Hypertens 28(2):183–194. https://doi.org/10.1097/MNH.0000000000000486
4. Winn MP, Conlon PJ, Lynn KL, Farrington MK, Creazzo T, Hawkins AF, Daskalakis N, Kwan SY, Ebersviller S, Burchette JL, Pericak-Vance MA, Howell DN, Vance JM, Rosenberg PB (2005) A mutation in the TRPC6 cation channel causes familial focal segmental glomerulosclerosis. Science 308(5729):1801–1804. https://doi.org/10.1126/science.1106215
5. Group KGW (2012) KDIGO clinical practice guideline for glomerulonephritis. Kidney Int 2:139–274
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2. Complete remission of tip lesion variant focal segmental glomerulosclerosis (FSGS) with the Janus Kinase (JAK) inhibitor tofacitinib;CEN Case Reports;2021-11-05
3. Degree of foot process effacement in patients with genetic focal segmental glomerulosclerosis: a single-center analysis and review of the literature;Scientific Reports;2021-06-08
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