Global DNA and protein interactomes of FLT1P1 (Fms-related tyrosine kinase 1 pseudogene 1) revealed its molecular regulatory functions associated with preeclampsia-Reference-Cited by-同舟云学术

登录注册会员服务联系我们

Global DNA and protein interactomes of FLT1P1 (Fms-related tyrosine kinase 1 pseudogene 1) revealed its molecular regulatory functions associated with preeclampsia

Published:2022-11-30 Issue:2 Volume:50 Page:1267-1279
ISSN:0301-4851
Container-title:Molecular Biology Reports
language:en
Short-container-title:Mol Biol Rep

Author:

Zhao Lu,Xin Siming,Wu Yunfei,Huang Shaofang,Xu Kangxiang,Xu Yuqi,Ruan Dong,Wu Bingqi,Chen Dong,He Xiaoju^ORCID

Funder

National Natural Science Foundation of China

Natural Science Foundation of Jiangxi Province

Science and Technology Project of Jiangxi Provincial Health Commission

Publisher

Springer Science and Business Media LLC

Subject

Genetics,Molecular Biology,General Medicine

Link

https://link.springer.com/content/pdf/10.1007/s11033-022-08070-0.pdf

Reference58 articles.

1. Rana S, Lemoine E, Granger JP, Karumanchi SA (2019) Preeclampsia: pathophysiology, challenges, and perspectives. Circ Res 124:1094–1112

2. Duckitt K, Harrington D (2005) Risk factors for pre-eclampsia at antenatal booking: systematic review of controlled studies. BMJ 330:565

3. Redman CW, Sargent IL (2005) Latest advances in understanding preeclampsia. Science 308:1592–1594

4. Romero R, Chaiworapongsa T (2013) Preeclampsia: a link between trophoblast dysregulation and an antiangiogenic state. J Clin Invest 123:2775–2777

5. Gray KJ, Kovacheva VP, Mirzakhani H, Bjonnes AC, Almoguera B, DeWan AT, Triche EW, Saftlas AF, Hoh J, Bodian DL (2018) Gene-centric analysis of preeclampsia identifies maternal association at PLEKHG1. Hypertension 72:408–416

同舟云学术

1.学者识别学者识别

2.学术分析学术分析

3.人才评估人才评估

"同舟云学术"是以全球学者为主线，采集、加工和组织学术论文而形成的新型学术文献查询和分析系统，可以对全球学者进行文献检索和人才价值评估。用户可以通过关注某些学科领域的顶尖人物而持续追踪该领域的学科进展和研究前沿。经过近期的数据扩容，当前同舟云学术共收录了国内外主流学术期刊6万余种，收集的期刊论文及会议论文总量共计约1.5亿篇，并以每天添加12000余篇中外论文的速度递增。我们也可以为用户提供个性化、定制化的学者数据。欢迎来电咨询！咨询电话：010-8811{复制后删除}0370

www.globalauthorid.com

TOP