Bombesin-like receptor 3 expression induced by bisphenol A is likely associated with reduced cell proliferation by inhibiting DNA synthesis and inducing inflammation in liver cells
Author:
Funder
Malaysia Research University Network (MRUN) Program Grant by Ministry of Higher Education Malaysia
Publisher
Springer Science and Business Media LLC
Link
https://link.springer.com/content/pdf/10.1007/s11033-023-09080-2.pdf
Reference52 articles.
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2. Bertoli S, Leone A, Battezzati A (2015) Human bisphenol A exposure and the “diabesity phenotype.” Dose Response 13(3):1559325815599173. https://doi.org/10.1177/1559325815599173
3. Ohore OE, Zhang S (2019) Endocrine disrupting effects of bisphenol A exposure and recent advances on its removal by water treatment systems: a review. Sci Afr 5:e00135. https://doi.org/10.1016/j.sciaf.2019.e00135
4. Peyre L, Rouimi P, de Sousa G, Héliès-Toussaint C, Carré B, Barcellini S, Chagnon MC, Rahmani R (2014) Comparative study of bisphenol A and its analogue bisphenol S on human hepatic cells: a focus on their potential involvement in nonalcoholic fatty liver disease. Food Chem Toxicol 70:9–18. https://doi.org/10.1016/j.fct.2014.04.011
5. Eweda SM, Newairy ASA, Abdou HM, Gaber AS (2020) Bisphenol A-induced oxidative damage in the hepatic and cardiac tissues of rats: the modulatory role of sesame lignans. Exp Ther Med 19(1):33–44. https://doi.org/10.3892/etm.2019.8193
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