The relationship between B-cell lymphoma 2, interleukin-1β, interleukin-17, and interleukin-33 and the development of diabetic nephropathy

Author:

Mahmoud Basant,Abdel-Moneim Adel,Negeem Zinab,Nabil AhmedORCID

Funder

Self funded

Publisher

Springer Science and Business Media LLC

Subject

Genetics,Molecular Biology,General Medicine

Reference46 articles.

1. Shahbazian H, Rezaii I (2013) Diabetic kidney disease; review of the current knowledge. J Ren Inj Prev 2(2):73–80

2. Gheith O, Farouk N, Nampoory N, Halim MA, Al-Otaibi T (2016) Diabetic kidney disease: worldwide difference of prevalence and risk factors. J Nephropharmacol 5(1):49–56

3. Taha H, Mahmoud HH, Soliman AS, Taha MM, Mohammed RA (2019) The Association between Highly Sensitive C-Reactive Protein and Interleukin-18 with Nephropathy in a Sample of Type 1 Diabetic Egyptian Patients. Med J Cairo Univ 87(4):2393–2402

4. Qin J, Peng Z, Yuan Q, Li Q et al (2019) AKF-PD alleviates diabetic nephropathy via blocking the RAGE/AGEs/NOX and PKC/NOX Pathways. Sci Rep 9(1):4407

5. Donate-Correa J, Ferri CM, Sánchez-Quintana F et al (2021) Inflammatory Cytokines in Diabetic Kidney Disease: Pathophysiologic and Therapeutic Implications. Front Med (Lausanne) 7:628289

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