CRISPR-Cas9 mediated genome editing of Huntington’s disease neurospheres
Author:
Funder
National Research Foundation of Korea
Publisher
Springer Science and Business Media LLC
Subject
Genetics,Molecular Biology,General Medicine
Link
https://link.springer.com/content/pdf/10.1007/s11033-022-08175-6.pdf
Reference49 articles.
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2. Andrew SE, Goldberg YP, Kremer B, Telenius H, Theilmann J, Adam S et al (1993) The relationship between trinucleotide (CAG) repeat length and clinical features of Huntington’s disease. Nat Genet 4(4):398–403. https://doi.org/10.1038/ng0893-398
3. Duyao M, Ambrose C, Myers R, Novelletto A, Persichetti F, Frontali M et al (1993) Trinucleotide repeat length instability and age of onset in Huntington’s disease. Nat Genet 4(4):387–392. https://doi.org/10.1038/ng0893-387
4. Diaz-Hernandez M, Torres-Peraza J, Salvatori-Abarca A, Moran MA, Gomez-Ramos P, Alberch J et al (2005) Full motor recovery despite striatal neuron loss and formation of irreversible amyloid-like inclusions in a conditional mouse model of Huntington’s disease. J Neurosci 25(42):9773–9781. https://doi.org/10.1523/JNEUROSCI.3183-05.2005
5. Yamamoto A, Lucas JJ, Hen R (2000) Reversal of neuropathology and motor dysfunction in a conditional model of Huntington’s disease. Cell 101(1):57–66. https://doi.org/10.1016/S0092-8674(00)80623-6
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