Canagliflozin ameliorates high glucose-induced apoptosis in NRK-52E cells via inhibiting oxidative stress and activating AMPK/mTOR-mediated autophagy-Reference-Cited by-同舟云学术

Canagliflozin ameliorates high glucose-induced apoptosis in NRK-52E cells via inhibiting oxidative stress and activating AMPK/mTOR-mediated autophagy

Published:2023-11-17 Issue:12 Volume:50 Page:10325-10337
ISSN:0301-4851
Container-title:Molecular Biology Reports
language:en
Short-container-title:Mol Biol Rep

Author:

Liu Hong^ORCID,Chen Weidong,Wan Sheng,Chen Ye,Fu Mengjing,Wang Zengsi,Xiong Fei,Zhang Yanmin

Funder

Scientific Research Project of Wuhan Municipal Health Commission

Publisher

Springer Science and Business Media LLC

Subject

Genetics,Molecular Biology,General Medicine

Link

https://link.springer.com/content/pdf/10.1007/s11033-023-08855-x.pdf

Reference43 articles.

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3. Thomas MC, Burns WC, Cooper ME (2005) Tubular changes in early diabetic nephropathy. Adv Chronic Kidney Dis 12:177–186. https://doi.org/10.1053/j.ackd.2005.01.008

4. Magri CJ, Fava S (2009) The role of tubular injury in diabetic nephropathy. Eur J Intern Med 20:551–555. https://doi.org/10.1016/j.ejim.2008.12.012

5. Mizushima N (2007) Autophagy: process and function. Genes Dev 21:2861–2873. https://doi.org/10.1101/gad.1599207

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