Role of Th22 cells in Helicobacter pylori-related gastritis and peptic ulcer diseases
Author:
Funder
Research Deputy of Shahrekord University of Medial Sciences
Publisher
Springer Science and Business Media LLC
Subject
Genetics,Molecular Biology,General Medicine
Link
http://link.springer.com/content/pdf/10.1007/s11033-019-05004-1.pdf
Reference34 articles.
1. Abolfathi M et al (2019) Associations of a NLRP3 rs10754558 polymorphism with Helicobacter pylori-infected patients with gastritis and peptic ulcer disease. Jundishapur J Microbiol 12:e88231. https://doi.org/10.5812/jjm.88231
2. Akdis M, Palomares O, van de Veen W, van Splunter M, Akdis CA (2012) TH17 and TH22 cells: a confusion of antimicrobial response with tissue inflammation versus protection. J Allergy Clin Immunol 129:1438–1449; quiz 1450–1431. https://doi.org/10.1016/j.jaci.2012.05.003
3. Andoh A et al (2005) Interleukin-22, a member of the IL-10 subfamily, induces inflammatory responses in colonic subepithelial myofibroblasts. Gastroenterology 129:969–984
4. Bagheri N, Azadegan-Dehkordi F, Rahimian G, Rafieian-Kopaei M, Shirzad H (2016) Role of regulatory T-cells in different clinical expressions of Helicobacter pylori infection. Arch Med Res 47:245–254. https://doi.org/10.1016/j.arcmed.2016.07.013
5. Bagheri N, Azadegan-Dehkordi F, Shirzad H, Rafieian-Kopaei M, Rahimian G, Razavi A (2015) The biological functions of IL-17 in different clinical expressions of Helicobacter pylori-infection. Microb Pathog 81:33–38. https://doi.org/10.1016/j.micpath.2015.03.010
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