Heat stress activates ER stress signals which suppress the heat shock response, an effect occurring preferentially in the cortex in rats-Reference-Cited by-同舟云学术

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Heat stress activates ER stress signals which suppress the heat shock response, an effect occurring preferentially in the cortex in rats

Published:2011-07-21 Issue:4 Volume:39 Page:3987-3993
ISSN:0301-4851
Container-title:Molecular Biology Reports
language:en
Short-container-title:Mol Biol Rep

Author:

Liu Yaohua,Sakamoto Hiroaki,Adachi Masaaki,Zhao Shiguang,Ukai Wataru,Hashimoto Eri,Hareyama Masato,Ishida Tadao,Imai Kohzoh,Shinomura Yasuhisa

Publisher

Springer Science and Business Media LLC

Subject

Genetics,Molecular Biology,General Medicine

Link

http://link.springer.com/content/pdf/10.1007/s11033-011-1179-2.pdf

Reference23 articles.

1. Morimoto RI (2008) Proteotoxic stress and inducible chaperone networks in neurodegenerative disease and aging. Genes Dev 22:1427–1438

2. McMillan DR, Xiao X, Shao L, Graves K, Benjamin IJ (1998) Targeted disruption of heat shock transcription factor 1 abolishes thermotolerance and protection against heat-inducible apoptosis. J Biol Chem 273:7523–7528

3. Luft JC, Benjamin IJ, Mestril R, Dix DJ (2001) Heat shock factor 1-mediated thermotolerance prevents cell death and results in G2/M cell cycle arrest. Cell Stress Chaperones 6:326–336

4. Zhang Y, Huang L, Zhang J, Moskophidis D, Mivechi NF (2002) Targeted disruption of hsf1 leads to lack of thermotolerance and defines tissue-specific regulation for stress-inducible Hsp molecular chaperones. J Cell Biochem 86:376–393

5. Kostova Z, Wolf DH (2003) For whom the bell tolls: protein quality control of the endoplasmic reticulum and the ubiquitin-proteasome connection. EMBO J 22:2309–2317

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