Mechanism of progression of diabetic kidney disease mediated by podocyte mitochondrial injury
Author:
Publisher
Springer Science and Business Media LLC
Subject
Genetics,Molecular Biology,General Medicine
Link
https://link.springer.com/content/pdf/10.1007/s11033-020-05749-0.pdf
Reference98 articles.
1. Lindblom R, Higgins G, Coughlan M, de Haan JB (2015) Targeting mitochondria and reactive oxygen species-driven pathogenesis in diabetic nephropathy. Rev Diabet Stud 12(1–2):134–156. https://doi.org/10.1900/RDS.2015.12.134
2. Schiffer TA, Friederich-Persson M (2017) Mitochondrial reactive oxygen species and kidney hypoxia in the development of diabetic nephropathy. Front Physiol 8:211. https://doi.org/10.3389/fphys.2017.00211
3. Lewis KUJB (2018) Update on diabetic nephropathy: core curriculum 2018. Am J Kidney Dis 71(6):884–895. https://doi.org/10.1053/j.ajkd.2017.10.026
4. Sagoo MK, Gnudi L (2018) Diabetic nephropathy: is there a role for oxidative stress? Free Radic Biol Med 116:50–63. https://doi.org/10.1016/j.freeradbiomed.2017.12.040
5. Ling-Feng Z, Xiao Y, Sun L (2019) A glimpse of the mechanisms related to renal fibrosis in diabetic nephropathy. Adv Exp Med Biol 1165:49–79
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