Digitalis cardiotoxicity: cellular calcium overload a possible mechanism-Reference-Cited by-同舟云学术

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Digitalis cardiotoxicity: cellular calcium overload a possible mechanism

Published:1989-11 Issue:6 Volume:84 Page:553-563
ISSN:0300-8428
Container-title:Basic Research in Cardiology
language:en
Short-container-title:Basic Res Cardiol

Author:

Khatter J. C.,Agbanyo M.,Navaratnam S.,Nero B.,Hoeschen R. J.

Publisher

Springer Science and Business Media LLC

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine,Physiology

Link

http://link.springer.com/content/pdf/10.1007/BF01906941.pdf

Reference46 articles.

1. Akera T (1971) Quantitative aspects of the interaction between ouabain and Na+, K+-ATPase in vitro. Biochem Biophys Acta 249:53–62

2. Allen DG, Blinks JR (1978) Calcium transients in aequorin-injected frog cardiac muscle. Nature 273:509–513

3. Bourdillon PVD, Pool-Wilson PA (1981) Effect of ischemia and reperfusion on calcium exchange and mechanical function in isolated rabbit myocardium. Cardiovasc Res 15:121–130

4. Carafoli E (1979) The calcium cycle of mitochondria. FEBS Lett 104:1–7

5. Carafoli E (1986) Membrane transport in the cellular homeostasis of calcium. J Cardiovasc Pharmacol 8:53–56

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1. Mitochondrial toxicity of cardiac drugs and its relevance to mitochondrial disorders;Expert Opinion on Drug Metabolism & Toxicology;2014-10-21

2. The H1–H2 domain of the α1 isoform of Na+–K+–ATPase is involved in ouabain toxicity in rat ventricular myocytes;Toxicology and Applied Pharmacology;2012-07

3. Prognostic Utility of Serum Potassium in Chronic Digoxin Toxicity;American Journal Cardiovascular Drugs;2011-06

4. Ouabain attenuates cardiotoxicity induced by other cardiac steroids;British Journal of Pharmacology;2010-03-01

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