Asymmetric diffusion into the postsynaptic neuron: An extremely efficient mechanism for removing excess GABA from synaptic clefts on the Deiters' neurone plasma membrane
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cellular and Molecular Neuroscience,General Medicine,Biochemistry
Link
http://link.springer.com/content/pdf/10.1007/BF00965338.pdf
Reference13 articles.
1. Cupello, A., andHyden, H. 1981. On the presence of met5-enkephalin receptors on the plasma membrane of Deiters' neurons and their modulation of GABA transport. J. Neurosci. Res. 6:579?583.
2. Early, S. L., Michaelis, E. K., andMertes, M. P. 1981. Pharmacological specificity of synaptosomal and synaptic membrane ?-aminobutyric acid (GABA) transport processes. Biochem. Pharmacol. 30:1105?1113.
3. Hyden, H., Cupello, A., andPalm, A. 1984. Increased binding of GABA to its postsynaptic carrier sites on the plasma membrane of Deiters' neurons after a learning experiment in rats. Brain. Res. 294:37?45.
4. Hyden, H., andLange, P. W. 1981. The effect of S100 protein on the plasma membrane function of neurons. Cellular and Molec. Neurobiol. 1:313?317.
5. Hyden, H., Lange, P. W., andLarsson, S. 1980. S-100 glia regulation of GABA transport across the nerve cell membrane. J. of Neurol. Sci. 45:303?316.
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