Sarcolemmal ion channels in dystrophin-deficient skeletal muscle fibres
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cell Biology,Biochemistry,Physiology
Link
http://link.springer.com/content/pdf/10.1007/s10974-006-9083-4.pdf
Reference34 articles.
1. Allard B, Lazdunski M, Rougier O (1995) Activation of ATP-dependent K+ channels by metabolic poisoning in adult mouse skeletal muscle: role of intracellular Mg2+ and pH. J Physiol 485:283–296
2. Allard B, Rougier O (1997) Similarity of ATP-dependent K+ channels in skeletal muscle fibres from normal and mutant mdx mice. J Physiol 498:319–325
3. Allard B, Couchoux H, Pouvreau S and Jacquemond V (2006) Sarcoplasmic reticulum Ca2+ -release and -depletion fail to affect sarcolemmal ion channel activity in mouse skeletal muscle. J Physiol, in press
4. Blake DJ, Weir A, Newey SE, Davies K (2002) Function and genetics of dystrophin and dystrophin-related proteins in muscle. Physiol Rev 82:291–329
5. Brennan C, Henderson LP (1993) Single channel properties of synaptic acetylcholine receptors in dystrophic fibers. Muscle Nerve 16:513–519
Cited by 23 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. Ion Channels of the Sarcolemma and Intracellular Organelles in Duchenne Muscular Dystrophy: A Role in the Dysregulation of Ion Homeostasis and a Possible Target for Therapy;International Journal of Molecular Sciences;2023-01-23
2. Drug Development and the Use of Induced Pluripotent Stem Cell-Derived Cardiomyocytes for Disease Modeling and Drug Toxicity Screening;International Journal of Molecular Sciences;2020-10-03
3. TRPCs: Influential Mediators in Skeletal Muscle;Cells;2020-04-01
4. Dystrophin restoration therapy improves both the reduced excitability and the force drop induced by lengthening contractions in dystrophic mdx skeletal muscle;Skeletal Muscle;2016-07-20
5. Disruption of action potential and calcium signaling properties in malformed myofibers from dystrophin-deficient mice;Physiological Reports;2015-04
1.学者识别学者识别
2.学术分析学术分析
3.人才评估人才评估
"同舟云学术"是以全球学者为主线,采集、加工和组织学术论文而形成的新型学术文献查询和分析系统,可以对全球学者进行文献检索和人才价值评估。用户可以通过关注某些学科领域的顶尖人物而持续追踪该领域的学科进展和研究前沿。经过近期的数据扩容,当前同舟云学术共收录了国内外主流学术期刊6万余种,收集的期刊论文及会议论文总量共计约1.5亿篇,并以每天添加12000余篇中外论文的速度递增。我们也可以为用户提供个性化、定制化的学者数据。欢迎来电咨询!咨询电话:010-8811{复制后删除}0370
www.globalauthorid.com
TOP
Copyright © 2019-2024 北京同舟云网络信息技术有限公司 京公网安备11010802033243号 京ICP备18003416号-3