The myosin-activated thin filament regulatory state, M − -open: a link to hypertrophic cardiomyopathy (HCM)
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cell Biology,Biochemistry,Physiology
Link
http://link.springer.com/content/pdf/10.1007/s10974-014-9383-z.pdf
Reference56 articles.
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3. Chang AN, Harada K, Ackerman MJ, Potter JD (2005) Functional consequences of hypertrophic and dilated cardiomyopathy-causing mutations in alpha-tropomyosin. J Biol Chem 280(40):34343–34349. doi: 10.1074/jbc.M505014200
4. Dong WJ, Cheung HC (1996) Calcium-induced conformational change in cardiac troponin C studied by fluorescence probes attached to Cys-84. Biochim Biophys Acta 1295(2):139–146
5. Dong WJ, Xing J, Villain M, Hellinger M, Robinson JM, Chandra M, Solaro RJ, Umeda PK, Cheung HC (1999) Conformation of the regulatory domain of cardiac muscle troponin C in its complex with cardiac troponin I. J Biol Chem 274(44):31382–31390
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