In situ study of myofibrils, mitochondria and bound creatine kinases in experimental cardiomyopathies

Author:

Veksler Vladimir,Ventura-Clapier Ren�e

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,General Medicine

Reference83 articles.

1. Savabi F, Kirsch A: Diabetic type of cardiomyopathy in food-restricted rats. Can J Physiol Pharmacol 70: 1040?1047, 1992

2. Kapelko VI, Veksler VI, Popovich MI: Cellular mechanisms of alterations in myocardial contractile function in experimental cardiomyopathies. Biomedical Science 1: 77?83, 1990

3. Kapelko VI, Veksler VI, Popovich MI, Ventura-Clapier R: Energy-linked functional alterations in experimental cardiomyopathies. Am J Physiol Suppl (Oct) 261: 39?44, 1991

4. Kapelko VI, Popovich MI, Veksler VI, Ventura-Clapier R, Khuchua ZA, Saks VA: Subcellular basis for increased diastolic stiffness in experimental cardiomyopathies. In: M Nagano, N Takeda, NS Dhalla (eds). The Cardiomyopathic Heart. Raven Press, Ltd, New York, 1994, pp 185?195

5. Veksler VI, Ventura-Clapier R, Lechene P, Vassort G: Functional state of myofibrils, mitochondria and bound creatine kinase in skinned ventricular fibers of cardiomyopathic hamsters. J Mol Cell Cardiol 20: 329?342, 1988

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