Transient expression of neuropeptide Y (NPY) immunoreactivity in the developing hamster paraventricular thalamic area is due to apoptosis
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cell Biology,Cellular and Molecular Neuroscience,General Medicine
Link
http://link.springer.com/content/pdf/10.1007/BF02151902.pdf
Reference38 articles.
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2. Arends, M. J., and Wyllie, A. H. (1990). Apoptosis: mechanisms and roles in pathology.Int. Rev. Exp. Pathol. 32:223–255.
3. Berger, B., Verney, C., Gaspar, P., and Febvert, A. (1985). Transient expression of tyrosine hydroxylase immunoreactivity in some neurons of the rat neocortex during postnatal development.Dev. Brain Res. 23:141–144.
4. Botchkina, G. I., and Morin, L. P. (1993). Development of the hamster serotonergic system: Cell groups and diencephalic projections.J. Comp. Neurol. 338:405–431.
5. Botchkina, G. I., and Morin, L. P. (1995). Organization of permament and transient NPY-IR neuron groups and fiber systems in the developing hamster diencephalon.J. Comp. Neurol. 357:573–602.
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