SS31 Ameliorates Sepsis-Induced Heart Injury by Inhibiting Oxidative Stress and Inflammation
Author:
Publisher
Springer Science and Business Media LLC
Subject
Immunology,Immunology and Allergy
Link
http://link.springer.com/content/pdf/10.1007/s10753-019-01081-3.pdf
Reference33 articles.
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3. Drosatos, K., A. Lymperopoulos, P.J. Kennel, N. Pollak, P.C. Schulze, and I.J. Goldberg. 2015. Pathophysiology of sepsis-related cardiac dysfunction: driven by inflammation, energy mismanagement, or both? Current Heart Failure Reports 12 (2): 130–140.
4. Liu, Y.C., M.M. Yu, S.T. Shou, and Y.F. Chai. 2017. Sepsis-induced cardiomyopathy: mechanisms and treatments. Frontiers in Immunology 8 (1): 1021.
5. Okuhara, Y., S. Yokoe, T. Iwasaku, A. Eguchi, K. Nishimura, W. Li, M. Oboshi, Y. Naito, T. Mano, M. Asahi, H. Okamura, T. Masuyama, and S. Hirotani. 2017. Interleukin-18 gene deletion protects against sepsis-induced cardiac dysfunction by inhibiting PP2A activity. International Journal of Cardiology 243: 396–403.
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