Neutrophil Extracellular Traps Induce Pyroptosis of Rheumatoid Arthritis Fibroblast-Like Synoviocytes via the NF-κB/Caspase 3/GSDME Pathway
Author:
Publisher
Springer Science and Business Media LLC
Subject
Immunology,Immunology and Allergy
Link
https://link.springer.com/content/pdf/10.1007/s10753-023-01951-x.pdf
Reference37 articles.
1. Nygaard, G., and G.S. Firestein. 2020. Restoring synovial homeostasis in rheumatoid arthritis by targeting fibroblast-like synoviocytes. Nature Reviews Rheumatology 16 (6): 316–333. https://doi.org/10.1038/s41584-020-0413-5.
2. Lillegraven, S., F.H. Prince, N.A. Shadick, V.P. Bykerk, B. Lu, M.L. Frits, et al. 2012. Remission and radiographic outcome in rheumatoid arthritis: application of the 2011 ACR/EULAR remission criteria in an observational cohort. Annals of the Rheumatic Diseases 71 (5): 681–686. https://doi.org/10.1136/ard.2011.154625.
3. Komatsu, N., and H. Takayanagi. 2022. Mechanisms of joint destruction in rheumatoid arthritis-immune cell-fibroblast-bone interactions. Nature Reviews Rheumatology 18 (7): 415–429. https://doi.org/10.1038/s41584-022-00793-5.
4. Bartok, B., and G.S. Firestein. 2010. Fibroblast-like synoviocytes: key effector cells in rheumatoid arthritis. Immunological Reviews 233 (1): 233–255. https://doi.org/10.1111/j.0105-2896.2009.00859.x.
5. Wu, Z., D. Ma, H. Yang, J. Gao, G. Zhang, K. Xu, et al. 2021. Fibroblast-like synoviocytes in rheumatoid arthritis: surface markers and phenotypes. International Immunopharmacology 93: 107392. https://doi.org/10.1016/j.intimp.2021.107392.
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