Role of the IL-23-T-bet/GATA3 Axis for the Pathogenesis of Ulcerative Colitis
Author:
Funder
KAKENHI
Publisher
Springer Science and Business Media LLC
Subject
Immunology,Immunology and Allergy
Link
https://link.springer.com/content/pdf/10.1007/s10753-020-01358-y.pdf
Reference30 articles.
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3. Xavier, R.J., and D.K. Podolsky. 2007. Unravelling the pathogenesis of inflammatory bowel disease. Nature. 448: 427–434.
4. Rosen, M.J., R. Karns, J.E. Vallance, R. Bezold, A. Waddell, M.H. Collins, Y. Haberman, P. Minar, R.N. Baldassano, J.S. Hyams, S.S. Baker, R. Kellermayer, J.D. Noe, A.M. Griffiths, J.R. Rosh, W.V. Crandall, M.B. Heyman, D.R. Mack, M.D. Kappelman, J. Markowitz, D.E. Moulton, N.S. Leleiko, T.D. Walters, S. Kugathasan, K.T. Wilson, S.P. Hogan, and L.A. Denson. 2017. Mucosal expression of type 2 and type 17 immune response genes distinguishes ulcerative colitis from colon-only Crohn’s disease in treatment-naive pediatric patients. Gastroenterology. 152: 1345–1357.
5. Hirahara, K., and T. Nakayama. 2016. CD4+ T-cell subsets in inflammatory diseases: beyond the Th1/Th2 paradigm. International Immunology 28: 163–171.
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