Mice Deficient in NOX2 Display Severe Thymic Atrophy, Lymphopenia, and Reduced Lymphopoiesis in a Zymosan-Induced Model of Systemic Inflammation
Author:
Funder
Japan Society for the Promotion of Science
Publisher
Springer Science and Business Media LLC
Subject
Immunology,Immunology and Allergy
Link
https://link.springer.com/content/pdf/10.1007/s10753-020-01342-6.pdf
Reference57 articles.
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2. Nauseef, W.M., and R.A. Clark. 2019. Intersecting stories of the phagocyte NADPH oxidase and chronic granulomatous disease. Methods Mol Biol 1982: 3–16. https://doi.org/10.1007/978-1-4939-9424-3_1.
3. Nunes, P., N. Demaurex, and M.C. Dinauer. 2013. Regulation of the NADPH oxidase and associated ion fluxes during phagocytosis. Traffic 14 (11): 1118–1131. https://doi.org/10.1111/tra.12115.
4. Arnold, D.E., and J.R. Heimall. 2017. A review of chronic granulomatous disease. Adv Ther 34 (12): 2543–2557. https://doi.org/10.1007/s12325-017-0636-2.
5. Dinauer, M.C. 2016. Primary immune deficiencies with defects in neutrophil function. Hematology Am Soc Hematol Educ Program 2016 (1): 43–50. https://doi.org/10.1182/asheducation-2016.1.43.
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1. Neutrophil metabolomics in severe COVID-19 reveal GAPDH as a suppressor of neutrophil extracellular trap formation;Nature Communications;2023-05-05
2. Zymosan Particle-Induced Hemodynamic, Cytokine and Blood Cell Changes in Pigs: An Innate Immune Stimulation Model with Relevance to Cytokine Storm Syndrome and Severe COVID-19;International Journal of Molecular Sciences;2023-01-06
3. Zymosan-induced leukocyte and cytokine changes in pigs: a new model for streamlined drug testing against severe COVID-19;2022-09-26
4. NOX2-Deficient Neutrophils Facilitate Joint Inflammation Through Higher Pro-Inflammatory and Weakened Immune Checkpoint Activities;Frontiers in Immunology;2021-09-07
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