Prolidase Could Act as a Diagnosis and Treatment Mediator in Lung Fibrosis-Reference-Cited by-同舟云学术

Prolidase Could Act as a Diagnosis and Treatment Mediator in Lung Fibrosis

Published:2012-06-21 Issue:5 Volume:35 Page:1747-1752
ISSN:0360-3997
Container-title:Inflammation
language:en
Short-container-title:Inflammation

Author:

Türkbeyler Ibrahim,Demir Tuncer,Pehlivan Yavuz,Kaplan Davut Sinan,Ceribasi Ali Osman,Orkmez Mustafa,Aksoy Nurten,Taysi Seyithan,Kisacik Bunyamin,Onat Ahmet Mesut

Publisher

Springer Science and Business Media LLC

Subject

Immunology,Immunology and Allergy

Link

http://link.springer.com/content/pdf/10.1007/s10753-012-9493-y.pdf

Reference32 articles.

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2. Gw, H., and K. Ar. 1995. Approaches to the treatment of pulmonary fibrosis. American Journal of Respiratory and Critical Care Medicine 151: 915–918.

3. Es, B., B. Ak, H. Jc, and R. Cr. 1996. Proteoglycan deposition in pulmonary fibrosis. American Journal of Respiratory and Critical Care Medicine 154: 1819–1828.

4. Palka, J.A., and J.M. Phang. 1997. Prolidase activity in fibroblasts is regulated by interaction of extracellular matrix with cell surface integrin receptors. Journal Of Cellular Biochemistry 67: 166–175.

5. Myara, I., C. Wolfrom, C. Charpentier, M. Gautier, and A. Lemonnier. 1984. Relationship between cell density and prolidase activity in human skin fibroblasts: effects of ascorbate and fructose. Biochimie 66(6): 445–449.

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