Guanylate-cyclase-mediated inhibition of cardiac I Ca by carbachol and sodium nitroprusside

Author:

Levi Renzo Cesare,Alloatti Giuseppe,Penna Claudia,Gallo Maria Pia

Publisher

Springer Science and Business Media LLC

Subject

Physiology (medical),Clinical Biochemistry,Physiology

Reference27 articles.

1. Balligand JL, Kelly RA, Marsden PA, Smith TW, Michel T (1993) Control of cardiac muscle cell function by endogenous nitric oxide signaling system. Proc Natl Acad Sci USA 90:347?351

2. Brown BS, Polson JB, Krzanowski JJ, Wiggins JR (1980) Influence of isoproterenol and methylisobutylxanthine on the contractile and cyclic nucleotide effects of methacholine in isolated rat atria. J Pharmacol Exp Ther 212:325?332

3. Burt JM, Spray DC (1988) Inotropic agents modulate gap junctional conductance between cardiac myocytes. Am J Physiol 254: H1206-H1210

4. Chinkers M, Garbers DL, Chang MS, Lowe DG, Chin H, Goeddel DV, Schulz S (1989) A membrane form of guanylate cyclase is an atrial natriuretic peptide receptor. Nature 338:78?83

5. Fischmeister R, Hartzell HC (1986) Mechanism of action of acetylcholine on calcium current in single cells from frog ventricle. J Physiol (Lond) 376:183?202

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