Hyper-SUMOylation of K+ Channels in Sudden Unexplained Death in Epilepsy: Isolation and Primary Culture of Dissociated Hippocampal Neurons from Newborn Mice for Subcellular Localization-Reference-Cited by-同舟云学术

Hyper-SUMOylation of K+ Channels in Sudden Unexplained Death in Epilepsy: Isolation and Primary Culture of Dissociated Hippocampal Neurons from Newborn Mice for Subcellular Localization

Published:2017-10-21 Issue: Volume: Page:63-71
ISSN:1064-3745
Container-title:Methods in Molecular Biology
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Author:

Chen Xu,Zhang Shanshan,Huang Jifang,Dong Wanying,Xiao Hui,Shao Huanjie,Cheng Jinke,Wu Hongmei,Qi Yitao

Publisher

Springer New York

Link

http://link.springer.com/content/pdf/10.1007/978-1-4939-7362-0_6

Reference15 articles.

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2. Brenner R, Wilcox KS (2012) Potassium channelopathies of epilepsy. In: Noebels JL et al (eds) Jasper’s basic mechanisms of the epilepsies. National Center for Biotechnology Information, Bethesda, MD

3. Yeh ET, Gong L, Kamitani T (2000) Ubiquitin-like proteins: new wines in new bottles. Gene 248(1-2):1–14

4. Kang X et al (2010) SUMO-specific protease 2 is essential for suppression of polycomb group protein-mediated gene silencing during embryonic development. Mol Cell 38(2):191–201

5. Qi Y et al (2014) An essential role of small ubiquitin-like modifier (SUMO)-specific Protease 2 in myostatin expression and myogenesis. J Biol Chem 289(6):3288–3293

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