Enhancement of fluoxetine-dependent increase of extracellular serotonin (5-HT) levels by (−)-pindolol, an antagonist at 5-HT1A receptors-Reference-Cited by-同舟云学术

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Enhancement of fluoxetine-dependent increase of extracellular serotonin (5-HT) levels by (−)-pindolol, an antagonist at 5-HT1A receptors

Published:1996-05 Issue:5 Volume:21 Page:557-562
ISSN:0364-3190
Container-title:Neurochemical Research
language:en
Short-container-title:Neurochem Res

Author:

Dreshfield Laura J.,Wong David T.,Perry Kenneth W.,Engleman Eric A.

Publisher

Springer Science and Business Media LLC

Subject

Cellular and Molecular Neuroscience,General Medicine,Biochemistry

Link

http://link.springer.com/content/pdf/10.1007/BF02527753.pdf

Reference33 articles.

1. Hjorth, S. 1993. Serotonin 5-HT1A autoreceptor blockade potentiates the ability of the 5-HT reuptake inhibitor citalopram to increase nerve terminal output of 5-HT in vivo: a microdialysis study. J. Neurochem. 60:776–779.

2. Invernizzi, R., Belli, S., and Samanin, R. 1992. Citalopram’s ability to increase extracellular concentrations of serotonin in the dorsal raphe prevents the drug’s effect in the frontal cortex. Brain Res. 584:322–324.

3. Gartside, S. E., Umbers, V., Hajós, M., and Sharp, T. 1995. Interaction between a selective 5-HT1A receptor antagonist and an SSRI in vivo: effects on 5-HT cell firing and extracellular 5-HT. Bri. J. Pharmacol. 115:1064–1070.

4. Hoyer, D., Engle, G., and Kalkman, H. O. 1985. Molecular pharmacology of 5-HT1 and 5-HT2 recognition sites in rat and pig brain membranes: radioligand binding studies with [3H]5-HT, [3H]8-OH-DPAT, (−)[125I]iodocyanopindolol,[3H]mesulergine and [3H]ketanserin. Eur. J. Pharmacol. 118:13–23.

5. Artigas, F., Perez, V., and Alvarez, E. 1994. Pindolol induces a rapid improvement of depressed patients treated with serotonin reuptake inhibitors. Arch. Gen. Psychiatry 51:248–251.

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