A refined diagnostic algorithm for polycythemia vera that incorporates mutation screening for JAK2V617F
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cancer Research,Oncology,Hematology
Link
http://link.springer.com/content/pdf/10.1007/s11899-006-0027-2.pdf
Reference68 articles.
1. Adamson JW, Fialkow PJ, Murphy S, et al.: Polycythemia vera: stem-cell and probable clonal origin of the disease. N Engl J Med 1976, 295:913–916.
2. Finazzi G, Caruso V, Marchioli R, et al.: Acute leukemia in polycythemia vera. An analysis of 1,638 patients enrolled in a prospective observational study. Blood 2005, 105:2664–2670. A very important source of information concerning hydroxyurea, regarding the lack of evidence for drug leukemogenicity.
3. Tefferi A, Gilliland DG: The JAK2 V617F tyrosine kinase mutation in myeloproliferative disorders: status report and immediate implications for disease classification and diagnosis. Mayo Clin Proc 2005, 80:947–958.
4. Jones AV, Kreil S, Zoi K, et al.: Widespread occurrence of the JAK2 V617F mutation in chronic myeloproliferative disorders. Blood 2005, 106:2162–2168. One of the first studies to reveal the occurrence of the JAK2V617F mutation in a diverse group of chronic myeloid disorders.
5. James C, Ugo V, Le Couedic JP, et al.: A unique clonal JAK2 mutation leading to constitutive signalling causes polycythaemia vera. Nature 2005, 434:1144–1148. A major discovery in polycythemia vera and related disorders.
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