Acetylcholine inhibits long-term hypoxia-induced apoptosis by suppressing the oxidative stress-mediated MAPKs activation as well as regulation of Bcl-2, c-IAPs, and caspase-3 in mouse embryonic stem cells-Reference-Cited by-同舟云学术

Acetylcholine inhibits long-term hypoxia-induced apoptosis by suppressing the oxidative stress-mediated MAPKs activation as well as regulation of Bcl-2, c-IAPs, and caspase-3 in mouse embryonic stem cells

Published:2007-11-30 Issue:2 Volume:13 Page:295-304
ISSN:1360-8185
Container-title:Apoptosis
language:en
Short-container-title:Apoptosis

Author:

Kim Min Hee,Kim Mi Ok,Heo Jung Sun,Kim Jin Sang,Han Ho Jae

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Biochemistry (medical),Cell Biology,Clinical Biochemistry,Pharmaceutical Science,Pharmacology

Link

http://link.springer.com/content/pdf/10.1007/s10495-007-0160-y.pdf

Reference44 articles.

1. Zhu LL, Wu LY, Yew DT et al (2005) Effects of hypoxia on the proliferation and differentiation of NSCs. Mol Neurobiol 31:231–242

2. Bunn HF, Poyton RO (1996) Oxygen sensing and molecular adaptation to hypoxia. Physiol Rev 76:839–885

3. Duranteau J, Chandel NS, Kulisz A et al (1998) Intracellular signaling by reactive oxygen species during hypoxia in cardiomyocytes. J Biol Chem 273:11619–11624

4. Vexler ZS, Ferriero DM (2001) Molecular and biochemical mechanisms of perinatal brain injury. Semin Neonatol 6:99–108

5. Millar TM, Phan V, Tibbles LA (2007) ROS generation in endothelial hypoxia and reoxygenation stimulates MAPkinase signaling and kinase-dependent neutrophil recruitment. Free Radic Biol Med 42:1165–1177

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