Cathepsin B is involved in the heat shock induced cardiomyocytes apoptosis as well as the anti-apoptosis effect of HSP-70-Reference-Cited by-同舟云学术

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Cathepsin B is involved in the heat shock induced cardiomyocytes apoptosis as well as the anti-apoptosis effect of HSP-70

Published:2014-09-13 Issue:11 Volume:19 Page:1571-1580
ISSN:1360-8185
Container-title:Apoptosis
language:en
Short-container-title:Apoptosis

Author:

Hsu Shu-Fen,Hsu Chuan-Chih,Cheng Bor-Chih,Lin Cheng-Hsien

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Biochemistry (medical),Cell Biology,Clinical Biochemistry,Pharmaceutical Science,Pharmacology

Link

http://link.springer.com/content/pdf/10.1007/s10495-014-1033-9.pdf

Reference24 articles.

1. Chen SH, Niu KC, Lin MT (2006) Cerebrovascular dysfunction is an attractive target for therapy in heat stroke. Clin Exp Pharmacol Physiol 33:663–672

2. Chen SH, Lin MT, Chang CP (2013) Ischemic and oxidative damage to the hypothalamus may be responsible for heat stroke. Curr Neuropharmacol 11:129–140

3. Qian L, Song X, Ren H, Gong J, Cheng S (2004) Mitochondrial mechanism of heat stress-induced injury in rat cardiomyocyte. Cell Stress Chaperones 9:281–293

4. Hsu SF, Chao CM, Huang WT, Lin MT, Cheng BC (2013) Attenuating heat-induced cellular autophagy, apoptosis, and damage in H9c2 cardiomyocytes by preinducing HSP70 with heat shock preconditioning. Int J Hyperth 29:239–247

5. Cheng BC, Chang CP, Tsay YG, Wu TF, Hsu CY, Lin MT (2008) Body cooling causes normalization of cardiac protein expression and function in a rat heatstroke model. J Proteome Res 7:4935–4945

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