Gene–diet-related factors of hyperglycaemia in postmenopausal women-Reference-Cited by-同舟云学术

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Gene–diet-related factors of hyperglycaemia in postmenopausal women

Published:2018-02-20 Issue:2 Volume:59 Page:169-177
ISSN:1234-1983
Container-title:Journal of Applied Genetics
language:en
Short-container-title:J Appl Genetics

Author:

Grygiel-Górniak Bogna,Kaczmarek Elżbieta,Mosor Maria,Przysławski Juliusz,Nowak Jerzy

Funder

Polish National Science Centre

Publisher

Springer Science and Business Media LLC

Subject

Genetics,General Medicine

Link

http://link.springer.com/article/10.1007/s13353-018-0434-9/fulltext.html

Reference43 articles.

1. Agulló-Ortuño MT, Albaladejo MD, Parra S, Rodríguez-Manotas M, Fenollar M, Ruíz-Espejo F et al (2002) Plasmatic homocysteine concentration and its relationship with complications associated to diabetes mellitus. Clin Chim Acta 326:105–112

2. Alberti KG, Eckel RH, Grundy SM, Zimmet PZ, Cleeman JI, Donato KA et al (2009) International Diabetes Federation Task Force on Epidemiology and Prevention; National Heart, Lung, and Blood Institute; American Heart Association; World Heart Federation; International Atherosclerosis Society; International Association for the Study of Obesity. Harmonizing the metabolic syndrome: a joint interim statement of the International Diabetes Federation Task Force on Epidemiology and Prevention; National Heart, Lung, and Blood Institute; American Heart Association; World Heart Federation; International Atherosclerosis Society; and International Association for the Study of Obesity. Circulation 120:1640–1645

3. Al-Maskari MY, Waly MI, Ali A, Al-Shuaibi YS, Ouhtit A (2012) Folate and vitamin B12 deficiency and hyperhomocysteinemia promote oxidative stress in adult type 2 diabetes. Nutrition 28:e23–e26

4. Altshuler D, Hirschhorn JN, Klannemark M, Lindgren CM, Vohl MC, Nemesh J et al (2000) The common PPARg Pro12Ala polymorphism is associated with decreased risk of type 2 diabetes. Nat Genet 26:76–80

5. Bell GI, Polonsky KS (2001) Diabetes mellitus and genetically programmed defects in beta-cell function. Nature 414:788–791

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