Meta-analysis of association between Arg326Gln (rs1503185) and Gln276Pro (rs1566734) polymorphisms of PTPRJ gene and cancer risk
Author:
Funder
Wroclaw Medical University
Publisher
Springer Science and Business Media LLC
Subject
Genetics,General Medicine
Link
http://link.springer.com/article/10.1007/s13353-019-00481-3/fulltext.html
Reference21 articles.
1. Aya-Bonilla C, Green MR, Camilleri E et al (2013) High-resolution loss of heterozygosity screening implicates PTPRJ as a potential tumor suppressor gene that affects susceptibility to non-Hodgkin’s lymphoma. Genes Chromosom Cancer. https://doi.org/10.1002/gcc.22044
2. Bilotta A, Dattilo V, D’Agostino S et al (2016) A novel splice variant of the protein tyrosine phosphatase PTPRJ that encodes for a soluble protein involved in angiogenesis. Oncotarget. https://doi.org/10.18632/oncotarget.14350
3. Fournier P, Dussault S, Fusco A et al (2016) Tyrosine phosphatase PTPRJ/DEP-1 is an essential promoter of vascular permeability, angiogenesis, and tumor progression. Cancer Res. https://doi.org/10.1158/0008-5472.CAN-16-1071
4. Iuliano R, Le PI, Cristofaro C et al (2004) The tyrosine phosphatase PTPRJ/DEP-1 genotype affects thyroid carcinogenesis. Oncogene 23:8432–8438. https://doi.org/10.1038/sj.onc.1207766
5. Iuliano R, Palmieri D, He H et al (2010) Role of PTPRJ genotype in papillary thyroid carcinoma risk. Endocr Relat Cancer. https://doi.org/10.1677/ERC-10-0143
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