JAK2V617F mutation in patients with β-thalassemia disease: prevalence and clinical characteristics
Author:
Funder
Thai Society of Hematology
Faculty of Medicine, Khon Kaen University
Publisher
Springer Science and Business Media LLC
Subject
Hematology,General Medicine
Link
https://link.springer.com/content/pdf/10.1007/s00277-021-04582-0.pdf
Reference11 articles.
1. Kralovics R, Passamonti F, Buser AS et al (2005) A gain-of-function mutation of JAK2 in myeloproliferative disorders. N Engl J Med 352:1779–1790. https://doi.org/10.1056/NEJMoa051113
2. Rivella S (2012) The role of ineffective erythropoiesis in non-transfusion-dependent thalassemia. Blood Rev 26(Suppl 1):S12-15. https://doi.org/10.1016/S0268-960X(12)70005-X
3. Taher A, Shammaa D, Bazarbachi A et al (2009) Absence of JAK2 V617F mutation in thalassemia intermedia patients. Mol Biol Rep 36:1555–1557. https://doi.org/10.1007/s11033-008-9350-0
4. Tahannejad Asadi Z, Yarahmadi R, Saki N et al (2019) Investigation of JAK2V617F mutation prevalence in patients with beta thalassemia major. Lab Med. https://doi.org/10.1093/labmed/lmz045
5. Vlachaki E, Kalogeridis A, Neokleous N et al (2012) Absence of JAK2V617F mutation in patients with beta-thalassemia major and thrombocytosis due to splenectomy. Mol Biol Rep 39:6101–6105. https://doi.org/10.1007/s11033-011-1425-7
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