Endothelial dysfunction biomarkers in sickle cell disease: is there a role for ADMA and PAI-1?
Author:
Funder
CAPES
CNPq
FAPEMIG
Publisher
Springer Science and Business Media LLC
Subject
Hematology,General Medicine
Link
https://link.springer.com/content/pdf/10.1007/s00277-021-04695-6.pdf
Reference59 articles.
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2. Mozeleski BM, Rubaish AA, Ali AA, Romero J (2018) Perspective: a novel prognostic for sickle cell disease. Saudi J Med Med Sci 6:133–136. https://doi.org/10.4103/sjmms.sjmms_107_18
3. Bunn HF (1997) Pathogenesis and treatment of sickle cell disease. N Engl J Med 337:762–769. https://doi.org/10.1056/NEJM199709113371107
4. Labie D, Pagnier J, Lapoumeroulie C, Rouabhi F, Dunda-Belkhodja O, Chardin P, Beldjord C, Wajcman H, Fabry ME, Nagel RL (1985) Common haplotype dependency of high G gamma-globin gene expression and high HbF levels in beta-thalassemia and sickle cell anemia patients. Proc Natl Acad Sci USA 82:2111–2114. https://doi.org/10.1073/pnas.82.7.2111
5. Alsultan A, Aleem A, Ghabbour H, AlGahtani FH, Al-Shehri A, Osman ME, Kurban K, Alsultan MS, Bahakim H, Al-Momen AM (2012) Sickle cell disease subphenotypes in patients from Southwestern Province of Saudi Arabia. J Pediatr Hematol Oncol 34:79–84. https://doi.org/10.1097/mph.0b013e3182422844
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