Contributions of bone marrow monocytes/macrophages in myeloproliferative neoplasms with JAK2V617F mutation-Reference-Cited by-同舟云学术

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Contributions of bone marrow monocytes/macrophages in myeloproliferative neoplasms with JAK2V617F mutation

Published:2023-05-26 Issue:7 Volume:102 Page:1745-1759
ISSN:0939-5555
Container-title:Annals of Hematology
language:en
Short-container-title:Ann Hematol

Author:

Fan Wenjuan,Cao Weijie,Shi Jianxiang,Gao Fengcai,Wang Meng,Xu Linping,Wang Fang,Li Yingmei,Guo Rong,Bian Zhilei,Li Wei^ORCID,Jiang Zhongxing,Ma Wang

Funder

Natural Science Foundation of Henan Province

Natural Science Foundation of China

China Postdoctoral Science Foundation

Key research and development and promotion of special projects in Henan Province

Henan Province Medical Science and Technology Research Project

Outstanding Youth Fund of Henan Province

Publisher

Springer Science and Business Media LLC

Subject

Hematology,General Medicine

Link

https://link.springer.com/content/pdf/10.1007/s00277-023-05284-5.pdf

Reference103 articles.

1. Tefferi A (2016) Myeloproliferative neoplasms: a decade of discoveries and treatment advances. Am J Hematol 91(1):50–58

2. Dameshek W (1951) Some speculations on the myeloproliferative syndromes. Blood 6(4):372–375

3. Tong J, Sun T, Ma S et al (2021) Hematopoietic stem cell heterogeneity is linked to the initiation and therapeutic response of myeloproliferative neoplasms. Cell Stem Cell 28(3):502-513.e6

4. Maekawa T, Kato S, Kawamura T et al (2019) Increased SLAMF7high monocytes in myelofibrosis patients harboring JAK2V617F provide a therapeutic target of elotuzumab. Blood 134(10):814–825

5. Lai HY, Brooks SA, Craver BM et al (2019) Defective negative regulation of Toll-like receptor signaling leads to excessive TNF-α in myeloproliferative neoplasm. Blood Adv 3(2):122–131

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