Integrative analyses reveal outcome-associated and targetable molecular partnerships between TP53, BRD4, TNFRSF10B, and CDKN1A in diffuse large B-cell lymphoma
Author:
Funder
Minnesota State University, Mankato
Publisher
Springer Science and Business Media LLC
Subject
Hematology,General Medicine
Link
https://link.springer.com/content/pdf/10.1007/s00277-023-05478-x.pdf
Reference65 articles.
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3. Fisher RI, Gaynor ER, Dahlberg S, Oken MM, Grogan TM, Mize EM et al (1993) Comparison of a standard regimen (CHOP) with three intensive chemotherapy regimens for advanced non-Hodgkin’s lymphoma. N Engl J Med 328:1002–1006. https://doi.org/10.1056/NEJM199304083281404
4. Tilly H, Morschhauser F, Sehn LH, Friedberg JW, Trněný M, Sharman JP et al (2022) Polatuzumab vedotin in previously untreated diffuse large B-cell lymphoma. N Engl J Med 386. https://doi.org/10.1056/nejmoa2115304
5. Crump M, Neelapu SS, Farooq U, Van Den Neste E, Kuruvilla J, Westin J et al (2017) Outcomes in refractory diffuse large B-cell lymphoma: results from the international SCHOLAR-1 study. Blood 130:1800–1808. https://doi.org/10.1182/blood-2017-03-769620
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1. Natural killer cell-associated prognosis model characterizes immune landscape and treatment efficacy of diffuse large B cell lymphoma;Cytokine;2024-10
2. Progress in deciphering the role of p53 in diffuse large B-cell lymphoma: mechanisms and therapeutic targets;American Journal of Cancer Research;2024
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