Targeting the Androgen Receptor in Breast Cancer-Reference-Cited by-同舟云学术

Targeting the Androgen Receptor in Breast Cancer

Published:2015-02 Issue:2 Volume:17 Page:
ISSN:1523-3790
Container-title:Current Oncology Reports
language:en
Short-container-title:Curr Oncol Rep

Author:

Chia KeeMing,O’Brien Megan,Brown Myles,Lim Elgene

Publisher

Springer Science and Business Media LLC

Subject

Oncology

Link

http://link.springer.com/content/pdf/10.1007/s11912-014-0427-8.pdf

Reference50 articles.

1. Park S, Koo JS, Kim MS, Park HS, Lee JS, Lee JS, et al. Androgen receptor expression is significantly associated with better outcomes in estrogen receptor-positive breast cancers. Ann Oncol: Off J Eur Soc Med Oncol / ESMO. 2011;22(8):1755–62.

2. Niemeier LA, Dabbs DJ, Beriwal S, Striebel JM, Bhargava R. Androgen receptor in breast cancer: expression in estrogen receptor-positive tumors and in estrogen receptor-negative tumors with apocrine differentiation. Mod Pathol : Off J U S Can Acad Pathol Inc. 2010;23(2):205–12.

3. Qi JP, Yang YL, Zhu H, Wang J, Jia Y, Liu N, et al. Expression of the androgen receptor and its correlation with molecular subtypes in 980 chinese breast cancer patients. Breast Cancer : Basic Clin Research. 2012;6:1–8.

4. Ni M, Chen Y, Lim E, Wimberly H, Bailey ST, Imai Y, et al. Targeting androgen receptor in estrogen receptor-negative breast cancer. Cancer Cell. 2011;20(1):119–31. Describes the enrichment of AR in ER-?HER2+ breast cancers and provides a mechanism by which AR signalling can activate HER2 signalling via Wnt and HER3. Impairment of AR-induced cell proliferation by either AR, Wnt or HER2 blockage has clinical implications.

5. Lehmann BD, Bauer JA, Chen X, Sanders ME, Chakravarthy AB, Shyr Y, et al. Identification of human triple-negative breast cancer subtypes and preclinical models for selection of targeted therapies. J Clin Invest. 2011;121(7):2750–67. Identifies six subtypes within the triple negative breast cancers including the AR(+) luminal androgen subtype characterized by a steriod hormone gene signature, which confers sensitivity to bicalutamide, and a high frequency of PIKCA mutations.

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