Molecular Signaling and Its Pathogenic Alterations in Neuromuscular Junction Formation and Maintenance
Author:
Publisher
Springer Japan
Link
http://link.springer.com/content/pdf/10.1007/978-4-431-55561-2_19
Reference98 articles.
1. Antolik C et al (2006) The tetratricopeptide repeat domains of rapsyn bind directly to cytoplasmic sequences of the muscle-specific kinase. Neuroscience 141(1):87–100. doi: 10.1016/j.neuroscience.2006.03.035
2. Arimura S et al (2014) DOK7 gene therapy benefits mouse models of diseases characterized by defects in the neuromuscular junction. Science 345(6203):1505–1508. doi: 10.1126/science.1250744
3. Beeson D et al (2006) Dok-7 mutations underlie a neuromuscular junction synaptopathy. Science 313(5795):1975–1978. doi: 10.1126/science.1130837
4. Belaya K et al (2012) Identification of DPAGT1 as a new gene in which mutations cause a congenital myasthenic syndrome. Ann N Y Acad Sci 1275:29–35. doi: 10.1111/j.1749-6632.2012.06790.x
5. Bergamin E et al (2010) The cytoplasmic adaptor protein Dok7 activates the receptor tyrosine kinase MuSK via dimerization. Mol Cell 39(1):100–109. doi: 10.1016/j.molcel.2010.06.007
1.学者识别学者识别
2.学术分析学术分析
3.人才评估人才评估
"同舟云学术"是以全球学者为主线，采集、加工和组织学术论文而形成的新型学术文献查询和分析系统，可以对全球学者进行文献检索和人才价值评估。用户可以通过关注某些学科领域的顶尖人物而持续追踪该领域的学科进展和研究前沿。经过近期的数据扩容，当前同舟云学术共收录了国内外主流学术期刊6万余种，收集的期刊论文及会议论文总量共计约1.5亿篇，并以每天添加12000余篇中外论文的速度递增。我们也可以为用户提供个性化、定制化的学者数据。欢迎来电咨询！咨询电话：010-8811{复制后删除}0370
www.globalauthorid.com
TOP
Copyright © 2019-2024 北京同舟云网络信息技术有限公司 京公网安备11010802033243号 京ICP备18003416号-3