GDF11, a target of miR-32-5p, suppresses high-glucose-induced mitochondrial dysfunction and apoptosis in HK-2 cells through PI3K/AKT signaling activation-Reference-Cited by-同舟云学术

GDF11, a target of miR-32-5p, suppresses high-glucose-induced mitochondrial dysfunction and apoptosis in HK-2 cells through PI3K/AKT signaling activation

Published:2023-02-07 Issue:7 Volume:55 Page:1767-1778
ISSN:1573-2584
Container-title:International Urology and Nephrology
language:en
Short-container-title:Int Urol Nephrol

Author:

Wang Hongjie^ORCID,Zhang Yunxia,Liu Huan,Li Shuang

Funder

Natural Science Foundation of Heilongjiang Province

Publisher

Springer Science and Business Media LLC

Subject

Urology,Nephrology

Link

https://link.springer.com/content/pdf/10.1007/s11255-023-03495-3.pdf

Reference39 articles.

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3. Pagliarini DJ, Calvo SE, Chang B et al (2008) A mitochondrial protein compendium elucidates complex I disease biology. Cell 134:112–123. https://doi.org/10.1016/j.cell.2008.06.016

4. Soltoff SP (1986) ATP and the regulation of renal cell function. Annu Rev Physiol 48:9–31. https://doi.org/10.1146/annurev.ph.48.030186.000301

5. Forbes JM, Thorburn DR (2018) Mitochondrial dysfunction in diabetic kidney disease. Nat Rev Nephrol 14:291–312. https://doi.org/10.1038/nrneph.2018.9

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