BOO induces fibrosis and EMT in urothelial cells which can be recapitulated in vitro through elevated storage and voiding pressure cycles
Author:
Funder
National Institute of Diabetes and Digestive and Kidney Diseases
National Science Foundation
National Institute of General Medical Sciences
Publisher
Springer Science and Business Media LLC
Subject
Urology,Nephrology
Link
https://link.springer.com/content/pdf/10.1007/s11255-021-02942-3.pdf
Reference46 articles.
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2. Irwin DE, Milsom I, Hunskaar S, Reilly K, Kopp Z, Herschorn S, Coyne K, Kelleher C, Hampel C, Artibani W (2006) Population-based survey of urinary incontinence, overactive bladder, and other lower urinary tract symptoms in five countries: results of the EPIC study. Eur Urol 50:1306–1315
3. Hughes FM, Sexton SJ, Jin H, Govada V, Purves JT (2017) Bladder fibrosis during outlet obstruction is triggered through the NLRP3 inflammasome and the production of IL-1β. Am J Physiol Physiol 313:F603–F610
4. Lepor H (2007) Alpha blockers for the treatment of benign prostatic hyperplasia. Rev Urol 9:181
5. Andersson K-E, Gratzke C (2007) Pharmacology of α1-adrenoceptor antagonists in the lower urinary tract and central nervous system. Nat Clin Pract Urol 4:368
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