Overexpression of TRIM3 protects against LPS-induced acute kidney injury via repressing IRF3 pathway and NLRP3 inflammasome
Author:
Publisher
Springer Science and Business Media LLC
Subject
Urology,Nephrology
Link
https://link.springer.com/content/pdf/10.1007/s11255-021-03017-z.pdf
Reference37 articles.
1. Levey AS, James MT (2017) Acute kidney injury. Ann Intern Med 167(9):ITC66–ITC80. https://doi.org/10.7326/AITC201711070
2. Acute FA, Injury K (2018) Acute kidney injury. Nurs Clin N Am 53(4):499–510. https://doi.org/10.1016/j.cnur.2018.07.001
3. Küllmar M, Meersch M (2019) Perioperative akute Nierenschädigung [perioperative acute kidney injury]. Anaesthesist 68(4):194–201. https://doi.org/10.1007/s00101-019-0556-4
4. Tang TT, Lv LL, Pan MM et al (2018) Hydroxychloroquine attenuates renal ischemia/reperfusion injury by inhibiting cathepsin mediated NLRP3 inflammasome activation. Cell Death Dis 9(3):351. https://doi.org/10.1038/s41419-018-0378-3
5. Giraldo MI, Hage A, van Tol S, Rajsbaum R (2020) TRIM proteins in host defense and viral pathogenesis. Curr Clin Microbiol Rep. https://doi.org/10.1007/s40588-020-00150-8
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3. Theacrine alleviates sepsis-induced acute kidney injury by repressing the activation of NLRP3/Caspase-1 inflammasome;PeerJ;2022-10-04
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