Regional citrate anticoagulation with continuous renal replacement therapy as a cause of hypercalcemia
Author:
Funder
National Institute of Diabetes and Digestive and Kidney Diseases
Publisher
Springer Science and Business Media LLC
Link
https://link.springer.com/content/pdf/10.1007/s11657-024-01434-y.pdf
Reference24 articles.
1. Dickens F (1941) The citric acid content of animal tissues, with reference to its occurrence in bone and tumour. Biochem J 35(8–9):1011–1023. https://doi.org/10.1042/bj0351011
2. Hu Y-Y, Rawal A, Schmidt-Rohr K (2010) Strongly bound citrate stabilizes the apatite nanocrystals in bone. Proc Natl Acad Sci USA 107(52):22425–22429. https://doi.org/10.1073/pnas.1009219107
3. Franklin RB, Chellaiah M, Zou J, Reynolds MA, Costello LC (2014) Evidence that osteoblasts are specialized citrate-producing cells that provide the citrate for incorporation into the structure of bone. TOBONEJ 6(1):1–8. https://doi.org/10.2174/1876525401406010001
4. Granchi D, Baldini N, Ulivieri FM, Caudarella R (2019) Role of citrate in pathophysiology and medical management of bone diseases. Nutrients 11(11):2576. https://doi.org/10.3390/nu11112576
5. Gaudry S, Palevsky PM, Dreyfuss D (2022) Extracorporeal kidney-replacement therapy for acute kidney injury. N Engl J Med 386(10):964–975. https://doi.org/10.1056/NEJMra2104090
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