Neutral effect of Glioma-associated oncogene-1 expression on survival in myelofibrosis
Author:
Publisher
Springer Science and Business Media LLC
Subject
General Medicine
Link
http://link.springer.com/content/pdf/10.1007/s00508-019-01572-1.pdf
Reference9 articles.
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2. Bhagwat N, Keller MD, Rampal RK, et al. Improved efficacy of combination of JAK2 and hedgehog inhibitors in myelofibrosis (abstract). Blood. 2013;122:666. https://doi.org/10.1182/blood-2012-10-461830. ASH 55th annual meeting.
3. Schneider RK, Mullally A, Dugourd A, et al. Gli1(+) mesenchymal stromal cells are a key driver of bone marrow fibrosis and an important cellular therapeutic target. Cell Stem Cell. 2017;20(6):785–800.e8.
4. Bose P, Alfayez M, Verstovsek S. New concepts of treatment for patients with myelofibrosis. Curr Treat Options Oncol. 2019;20(1):5.
5. Gerds AT, Tauchi T, Ritchie E, et al. Phase 1/2 Trial of Glasdegib in Patients with Primary or Secondary Myelofibrosis Previously Treated with Ruxolitinib. Leuk Res. 2019;79:38–44. https://doi.org/10.1016/j.leukres.2019.02.012.
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1. The Role of Mutated Calreticulin in the Pathogenesis of BCR-ABL1-Negative Myeloproliferative Neoplasms;International Journal of Molecular Sciences;2024-09-12
2. Inflammation and bone marrow fibrosis: novel immunotherapeutic targets;Current Opinion in Hematology;2023-08-02
3. GLI1 activates pro-fibrotic pathways in myelofibrosis fibrocytes;Cell Death & Disease;2022-05-20
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