Antigen stimulation of human pulmonary smooth muscle: An in vitro model of inflammation

Author:

Gorenne I.,Labat C.,Gascard J. P.,Brink C.

Publisher

Springer Science and Business Media LLC

Subject

Health, Toxicology and Mutagenesis,Cell Biology,Toxicology

Reference10 articles.

1. Aurer J, Lewis PA. The physiology of the immediate reaction of anaphylaxis in the guinea-pig. J Exp Med. 1910; 12: 151?75.

2. Björck T, Dahlén S-E. Leukotrienes and histamine mediate IgE-dependent contractions of human bronchi: pharmacological evidence obtained with tissues from asthmatic and non-asthmatic subjects. Pulmonary Pharmacol. 1993; 6: 87?96.

3. Buckner CK, Krell RB, Laravuso RB et al. Pharmacological evidence that human intralobar airways do not contain different receptors that mediate contractions to leukotriene C4 and D4. J Pharmacol Exp Ther. 1986; 237: 558?62.

4. Gorenne I, Labat C, Gascard JP et al. (R)-2[4-(Quinolin-2-yl-methoxy)phenyl]-2-cyclopentyl acetic acid (BAY x1005), a potent leukotriene synthesis inhibitor: effects on anti-IgE challenge in human airways. J Pharmacol Exp Ther. 1994a;268:868?72.

5. Gorenne I, Labat C, Norel X et al. Anti-IgE response in human airways: relative contribution of inflammatory mediators. Mediators Inflamm. 1994b;3:359?63.

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3. Leukotrienes and the Pulmonary Vascular Bed;Advances in Experimental Medicine and Biology;2002

4. Functional Studies of Leukotriene Receptors in Vascular Tissues;American Journal of Respiratory and Critical Care Medicine;2000-02

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