MicroRNA-92a in Cardiovascular Disease: An Insufficiently Explored and Controversial Research Area
Author:
Publisher
Springer Science and Business Media LLC
Subject
Pharmacology (medical),Cardiology and Cardiovascular Medicine,Pharmacology,General Medicine
Link
https://link.springer.com/content/pdf/10.1007/s10557-023-07533-1.pdf
Reference5 articles.
1. Wu Q, Wang H, He F, et al. Depletion of microRNA-92a enhances the role of sevoflurane treatment in reducing myocardial ischemia–reperfusion injury by upregulating KLF4. Cardiovasc Drugs Ther. 2022. https://doi.org/10.1007/s10557-021-07303-x.
2. Hinkel R, Penzkofer D, Zühlke S, et al. Inhibition of microRNA-92a protects against ischemia/reperfusion injury in a large-animal model. Circulation. 2013;128(10):1066–75.
3. Zhang B, Zhou M, Li C, et al. MicroRNA-92a inhibition attenuates hypoxia/reoxygenation-induced myocardiocyte apoptosis by targeting Smad7. PLoS One. 2014;9(6):e100298.
4. Jiang F, Zhang B, Zhang X, et al. miRNA-92a inhibits vascular smooth muscle cell phenotypic modulation and may help prevent in-stent restenosis. Mol Med Rep. 2023;27(2):40.
5. Ma M, Li H, Yin S, Lin T, Song T. Overexpression of miR-92a attenuates kidney ischemia-reperfusion injury and improves kidney preservation by inhibiting MEK4/JNK1-related autophagy. Cell Mol Biol Lett. 2023;28(1):20.
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