Winding Down the Storm: Could Empagliflozin’s Ion-Dependent Mechanisms Tackle the Ventricular Arrhythmia Burden in Chronic Chagas Cardiomyopathy?
Author:
Publisher
Springer Science and Business Media LLC
Subject
Pharmacology (medical),Cardiology and Cardiovascular Medicine,Pharmacology,General Medicine
Link
https://link.springer.com/content/pdf/10.1007/s10557-023-07510-8.pdf
Reference5 articles.
1. Peng X, Li L, Lin R, et al. Empagliflozin ameliorates ouabain-induced Na+ and Ca2+ dysregulations in ventricular myocytes in an Na+-dependent manner. Cardiovasc Drugs Ther. 2023;37:461–9.
2. Nunes MCP, Beaton A, Acquatella H, et al. Chagas cardiomyopathy: an update of current clinical knowledge and management: a scientific statement from the American Heart Association. Circulation. 2018;138:e169–209.
3. Bögeholz N, Pauls P, Bauer BK, et al. Overexpression of the Na+ /Ca2+ exchanger influences ouabain-mediated spontaneous Ca2+ activity but not positive inotropy. Fundam Clin Pharmacol. 2019;33:43–51.
4. Li H-L, Lip GYH, Feng Q, et al. Sodium-glucose cotransporter 2 inhibitors (SGLT2i) and cardiac arrhythmias: a systematic review and meta-analysis. Cardiovasc Diabetol. 2021;20:100.
5. Santos-Miranda A, Joviano-Santos JV, Sarmento JO, et al. A novel substrate for arrhythmias in Chagas disease. PLoS Negl Trop Dis. 2021;15:e0009421.
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