MicroRNA-145 in Cardiovascular Disease: an Insufficiently Explored and Controversial Research Area
Author:
Publisher
Springer Science and Business Media LLC
Subject
Pharmacology (medical),Cardiology and Cardiovascular Medicine,Pharmacology,General Medicine
Link
https://link.springer.com/content/pdf/10.1007/s10557-022-07388-y.pdf
Reference5 articles.
1. Liu Y, Hu J, Wang W, Wang Q. MircroRNA-145 attenuates cardiac fibrosis via regulating mitogen-activated protein kinase kinase kinase 3. Cardiovasc Drugs Ther. 2022. https://doi.org/10.1007/s10557-021-07312-w.
2. Song HF, He S, Li SH, et al. Knock-out of MicroRNA 145 impairs cardiac fibroblast function and wound healing post-myocardial infarction. J Cell Mol Med. 2020;24(16):9409–19.
3. Ogawa K, Noda A, Ueda J, et al. Forced expression of miR-143 and -145 in cardiomyocytes induces cardiomyopathy with a reductive redox shift. Cell Mol Biol Lett. 2020;25:40. Published 2020 Aug 24.
4. Thomas S, Manivannan S, Sawant D, et al. miR-145 transgenic mice develop cardiopulmonary complications leading to postnatal death. Physiol Rep. 2021;9(17):e15013.
5. Ogorodnikova N, Arenz C. MicroRNA-145-targeted drug and its preventive effect on pulmonary arterial hypertension (patent WO2012153135 A1). Expert Opin Ther Pat. 2015;25(6):723–7.
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