Nitric oxide and prostaglandin as mediators in the pathogenesis of hyperkinetic circulatory state in a model of endotoxemia-induced portal hypertension

Author:

Rizvi Moattar Raza,Tauseef Mohammad,Shahid Mohd,Babbar Rashmi,Fahim Mohammad,Sakhuja Puja,Sarin Shiv Kumar

Publisher

Springer Science and Business Media LLC

Subject

Hepatology

Reference42 articles.

1. Wiest R. Splanchnic and systemic vasodilation: the experimental models. J Clin Gastroenterol 2007;41(Suppl 3):S272–287

2. Abraldes JG, Pasarín M, García-Pagán JC. Animal models of portal hypertension. World J Gastroenterol 2006;12(41):6577–6584

3. Kiel JW, Pitts L, Benoit JN, Granger DN, Shepherd AP. Reduced vascular sensitivity to norepinephrine in portal hypertensive rats. Am J Physiol 1985;248:G192–G195

4. Gadano AC, Sogni P, Heller J, et al. Vascular nitric oxide production during the development of two experimental models of portal hypertension. J Hepatol 1999;30(5):896–903

5. Cahill PA, Foster CL, Redmond EM, Gingalewski C, Wu Y, Sitzmann JV. Enhanced nitric oxide synthase activity in portal hypertensive rabbits. Hepatology. 1995;22:598–606.

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