Hyperdynamic circulation in cirrhosis: a novel gutsy central neuronal mechanism
Author:
Publisher
Springer Science and Business Media LLC
Subject
Hepatology
Link
https://link.springer.com/content/pdf/10.1007/s12072-023-10514-z.pdf
Reference10 articles.
1. Bernardi M, Moreau R, Angeli P, Schnabl B, Arroyo V. Mechanisms of decompensation and organ failure in cirrhosis: From peripheral arterial vasodilation to systemic inflammation hypothesis. J Hepatol. 2015;63:1272–1284
2. Schrier RW, Arroyo V, Bernardi M, Epstein M, Henriksen JH, Rodes J. Peripheral arterial vasodilation hypothesis: a proposal for the initiation of renal sodium and water retention in cirrhosis. Hepatology. 1988;8:1151–1157
3. Angeli P, Villanueva C, Francoz C, Mookerjee R, Trebicka J, Krag A, et al. European Association for the Study of the Liver (EASL) Clinical Practice Guidelines for the management of patients with decompensated cirrhosis. Journal of hepatology. 2018; 69(2), 406–460.
4. Liu H, Schuelert N, McDougall JJ, Lee SS. Central neural activation of hyperdynamic circulation in portal hypertensive rats depends on vagal afferent nerves. Gut. 2008;57:966–973
5. Song D, Liu H, Sharkey KA, Lee SS. Hyperdynamic circulation in portal-hypertensive rats is dependent on central c-fos gene expression. Hepatology. 2002;35:159–166
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