Construction of Lentiviral Vector for miR-217 Overexpression and Knockdown and Its Effect on CML-Reference-Cited by-同舟云学术

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Construction of Lentiviral Vector for miR-217 Overexpression and Knockdown and Its Effect on CML

Published:2022-12-10 Issue: Volume: Page:
ISSN:1073-6085
Container-title:Molecular Biotechnology
language:en
Short-container-title:Mol Biotechnol

Author:

Yang Xin,Jiang Huihui,Sun Chengming^ORCID

Funder

major scientific and technological innovation projects of the Shandong Province of China

Yantai Science and Technology Plan Project of China

Science and Technology Plan Project of Wenzhou, China

Publisher

Springer Science and Business Media LLC

Subject

Molecular Biology,Applied Microbiology and Biotechnology,Biochemistry,Bioengineering,Biotechnology

Link

https://link.springer.com/content/pdf/10.1007/s12033-022-00615-9.pdf

Reference31 articles.

1. Xu, J., Wu, M., Sun, Y., Zhao, H., Wang, Y., & Gao, J. (2020). Identifying dysregulated lncRNA-associated ceRNA network biomarkers in CML based on dynamical network biomarkers. BioMed Research International. https://doi.org/10.1155/2020/5189549

2. Vener, C., Banzi, R., Ambrogi, F., Ferrero, A., Saglio, G., Pravettoni, G., & Sant, M. (2020). First-line imatinib vs second- and third-generation TKIs for chronic-phase CML: A systematic review and meta-analysis. Blood Advances, 4(12), 2723–2735. https://doi.org/10.1182/bloodadvances.2019001329

3. Gustafson, D., Fish, J. E., Lipton, J. H., & Aghel, N. (2020). Mechanisms of cardiovascular toxicity of BCR-ABL1 tyrosine kinase inhibitors in chronic myelogenous leukemia. Current Hematologic Malignancy Reports, 15(1), 20–30. https://doi.org/10.1007/s11899-020-00560-x

4. Wang, N., Mi, M., Wei, X., & Sun, C. (2020). Circadian clock gene Period2 suppresses human chronic myeloid leukemia cell proliferation. Experimental and Therapeutic Medicine, 20(6), 147. https://doi.org/10.3892/etm.2020.9276

5. Fu, L., Pelicano, H., Liu, J., Huang, P., & Lee, C. (2002). The circadian gene Period2 plays an important role in tumor suppression and DNA damage response in vivo. Cell, 111(1), 41–50. https://doi.org/10.1016/s0092-8674(02)00961-3

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