Cerebral [18F]AIF-FAPI-42-Based PET Imaging of Fibroblast Activation Protein for Non-invasive Quantification of Fibrosis After Ischemic Stroke-Reference-Cited by-同舟云学术

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Cerebral [18F]AIF-FAPI-42-Based PET Imaging of Fibroblast Activation Protein for Non-invasive Quantification of Fibrosis After Ischemic Stroke

Published:2024-06-28 Issue: Volume: Page:
ISSN:1868-4483
Container-title:Translational Stroke Research
language:en
Short-container-title:Transl. Stroke Res.

Author:

Tang Peipei,Liu Yang,Peng Simin,Cai Zhikai,Tang Ganghua,Zhou Zhou,Hu Kongzhen,Zhong Yuhua

Funder

Science and Technology Projects in Guangzhou

National Natural Science Foundation of China

Basic and Applied Basic Research Foundation of Guangdong Province

National Isotope Engineering Research Center of China

Publisher

Springer Science and Business Media LLC

Link

https://link.springer.com/content/pdf/10.1007/s12975-024-01269-2.pdf

Reference40 articles.

1. Qin C, Yang S, Chu YH, et al. Signaling pathways involved in ischemic stroke: molecular mechanisms and therapeutic interventions. Signal Transduct Target Ther. 2022;7:215.

2. Dirnagl U, Iadecola C, Moskowitz MA. Pathobiology of ischaemic stroke: an integrated view. Trends Neurosci. 1999;22:391–7.

3. GBD 2019 Stroke Collaborators. Global, regional, and national burden of stroke and its risk factors, 1990–2019: a systematic analysis for the Global Burden of Disease Study 2019. Lancet Neurol. 2021;20:795–820.

4. Campbell BCV, De Silva DA, Macleod MR, et al. Ischaemic stroke. Nat Rev Dis Primers. 2019;5:70.

5. Wieloch T, Nikolich K. Mechanisms of neural plasticity following brain injury. Curr Opin Neurobiol. 2006;16:258–64.

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