Presence of BPIFB1 in saliva from non-obese diabetic mice
Author:
Funder
Grants-in-aids for Scientific Research from JSPS
Publisher
Springer Science and Business Media LLC
Subject
General Dentistry
Link
http://link.springer.com/article/10.1007/s10266-017-0312-7/fulltext.html
Reference29 articles.
1. Harada M. Immune disturbance and pathogenesis of non-obese diabetes-prone (NOD) mice. Exp Clin Endocrinol. 1987;89(3):251–8.
2. Hattori M, Buse JB, Jackson RA, Glimcher L, Dorf ME, Minami M, Makino S, Moriwaki K, Kuzuya H, Imura H, et al. The NOD mouse: recessive diabetogenic gene in the major histocompatibility complex. Science. 1986;231(4739):733–5.
3. Driver JP, Serreze DV, Chen YG. Mouse models for the study of autoimmune type 1 diabetes: a NOD to similarities and differences to human disease. Semin Immunopathol. 2011;33(1):67–87.
4. Nashida T, Yoshie S, Haga-Tsujimura M, Imai A, Shimomura H. Atrophy of myoepithelial cells in parotid glands of diabetic mice; detection using skeletal muscle actin, a novel marker. FEBS Open Bio. 2013;3:130–4.
5. Fukushima T, Nashida T, Haga-Tsujimura M, Mataga I. Chitinase expression in parotid glands of non-obese diabetic mice. Oral Dis. 2012;18(5):506–12.
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