Histone-deacetylase-inhibitory effects of periodontopathic-bacterial metabolites induce human gingival epithelial Ca9-22 cell death
Author:
Funder
Japan Society for the Promotion of Science
School of Dentistry, Nihon University
Dental Research Center
Publisher
Springer Science and Business Media LLC
Subject
General Dentistry
Link
https://link.springer.com/content/pdf/10.1007/s10266-022-00775-9.pdf
Reference23 articles.
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2. Tsuda H, Ochiai K, Suzuki N, Otsuka K. Butyrate, a bacterial metabolite, induces apoptosis and autophagic cell death in gingival epithelial cells. J Periodontal Res. 2010;45(5):626–34.
3. Evans M, Murofushi T, Tsuda H, Mikami Y, Zhao N, Ochiai K, et al. Combined effects of starvation and butyrate on autophagy-dependent gingival epithelial cell death. J Periodontal Res. 2017;52(3):522–31.
4. Fujiwara Y, Murofushi T, Koshi R, Mikami Y, Tsuda H. Reactive oxygen species-dependent release of damage-associated molecular patterns from human gingival epithelial Ca9-22 cells during butyrate or propionate exposure. J Oral Sci. 2021;63(2):195–7.
5. Kumar V, Thakur JK, Prasad M. Histone acetylation dynamics regulating plant development and stress responses. Cell Mol Life Sci. 2021;78(10):4467–86.
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