Substance P failed to reverse dextran sulfate sodium-induced murine colitis mediated by mitochondrial dysfunction: implications in ulcerative colitis
Author:
Funder
DST-SERB
Publisher
Springer Science and Business Media LLC
Subject
Agricultural and Biological Sciences (miscellaneous),Environmental Science (miscellaneous),Biotechnology
Link
https://link.springer.com/content/pdf/10.1007/s13205-021-02755-2.pdf
Reference50 articles.
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3. Bar F, Bochmann W, Widok A et al (2013) Mitochondrial gene polymorphisms that protect mice from colitis. Gastroenterol 145:1055–1063. https://doi.org/10.1053/j.gastro.2013.07.015
4. Berridge MV, Tan AS (1993) Characterization of the cellular reduction of 3-(4,5-dimethylthiazol-2-yl)-2,5-diphenyltetrazolium bromide (MTT): subcellular localization, substrate dependence, and involvement of mitochondrial electron transport in MTT reduction. Arch Biochem Biophys 303:474–482. https://doi.org/10.1006/abbi.1993.1311
5. Block ML, Li G, Qin L et al (2006) Potent regulation of microglia-derived oxidative stress and dopaminergic neuron survival: substance P vs. dynorphin. FASEB J 20:251–258. https://doi.org/10.1096/fj.05-4553com
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